首页/数据集/Supplementary Table S2 from Candidate Antimetastasis Drugs Suppress the Metastatic Capacity of Breast Cancer Cells by Reducing Membrane Fluidity

Supplementary Table S2 from Candidate Antimetastasis Drugs Suppress the Metastatic Capacity of Breast Cancer Cells by Reducing Membrane Fluidity

Figshare2016-04-01 更新2026-04-28 收录

抗肿瘤转移药物

膜流动性调控

数据链接：

https://figshare.com/articles/dataset/Supplementary_Table_S2_from_Candidate_Antimetastasis_Drugs_Suppress_the_Metastatic_Capacity_of_Breast_Cancer_Cells_by_Reducing_Membrane_Fluidity/22408592 数据链接链接失效反馈

官方服务：

资源简介：

Connectivity Map results.

应用场景：

创建时间：

2016-04-01

相关数据集

化学力编程组装核酸合成及性能调控数据集

核酸研究

抗肿瘤转移药物

本数据集主要面向抗肿瘤转移纳米药物的精准设计与合成研究需求建设，源于国家重点研发计划“纳米科技”专项课题一的研究工作。数据产生于福州大学及国家纳米科学中心的高端仪器分析平台，依托核磁共振（NMR）、高分辨质谱（HR-MS）、透射电镜（TEM）及分子动力学模拟工作站等设备采集。数据集主要记录了基于氰基介导、氟效应及硫醇交换等“化学力编程”策略构建的核酸纳米药物的全生命周期数据。内容涵盖关键功能分子的

国家基础学科公共科学数据中心150

The adiponectin receptor AdipoR2 and its Caenorhabditis elegans homolog PAQR-2 prevent membrane rigidification by exogenous saturated fatty acids

膜流动性调控

脂肪酸代谢毒性

Dietary fatty acids can be incorporated directly into phospholipids. This poses a specific challenge to cellular membranes since their composition, hence properties, could greatly vary with different

NIAID Data Ecosystem30

YK-4-279 Inhibits ERG and ETV1 Mediated Prostate Cancer Cell Invasion

前列腺癌靶向治疗

抗肿瘤转移药物

BackgroundGenomic rearrangements involving the ETS family of transcription factors occur in 40–70% of prostate cancer cases. ERG and ETV1 are the most common ETS members observed in these genetic alte

Figshare2016-01-18 更新30

The Tetraindole SK228 Reverses the Epithelial-to-Mesenchymal Transition of Breast Cancer Cells by Up-Regulating Members of the miR-200 Family

抗肿瘤转移药物

上皮-间质转化机制

The results of recent studies have shown that metastasis, the most common malignancy and primary cause of mortality promoted by breast cancer in women, is associated with the epithelial-to-mesenchymal

NIAID Data Ecosystem80

Membrane physical state controls the signaling mechanism of the heat shock response in Synechocystis PCC 6803: Identification of hsp17 as a “fluidity gene”

热休克反应

膜流动性调控

The fluidity of Synechocystis membranes was adjusted in vivo by temperature acclimation, addition of fluidizer agent benzyl alcohol, or catalytic lipid hydrogenation specific to plasma membranes. The

PubMed Central1998-03-31 更新30