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Additional file 2 of Relapse-specific genetic patterns in the exomic mutational landscape in acute myeloid leukemia

NIAID Data Ecosystem2026-05-02 收录

急性髓系白血病基因组学

复发特异性突变

数据链接：

https://figshare.com/articles/dataset/Additional_file_2_of_Relapse-specific_genetic_patterns_in_the_exomic_mutational_landscape_in_acute_myeloid_leukemia/28666124 数据链接链接失效反馈

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资源简介：

Supplementary Material 2

应用场景：

创建时间：

2025-03-25

急性髓系白血病基因组学

CEBPA基因

Acute myeloid leukaemia (AML) is a heterogeneous haematological malignancy caused by mutations in genes encoding transcriptional and epigenetic regulators together with signalling genes. It is charact

NIAID Data Ecosystem70

35549e9c-f1f9-4722-8181-ec8c3cd8fbbf - samples

急性髓系白血病基因组学

复发监测

Whole genome sequencing of AML blood or bone marrow at presentation and remission for 5 patients. Relapse samples are included for 2 patients, totaling 12 WGS BAM files.EGA dataset EGAD00001005120

NIAID Data Ecosystem70

Leucegene: AML sequencing (part 1)

急性髓系白血病基因组学

RNA测序分析

RNA sequencing of human leukemia Overall design: The goals of this project are to obtain a comprehensive study of mutations and gene expression in human acute myeloid leukemia (AML). Methods: AML cell

NIAID Data Ecosystem40

Next Generation Sequencing platform for targeted Personalized Therapy of Leukemia - Acute myeloid leukemia

急性髓系白血病基因组学

精准医疗生物标志物

The advent of high-throughput next generation sequencing (NGS) technologies that are revolutionizing genomics and transcriptomics by providing a single base resolution tool for a unified deep analysis

NIAID Data Ecosystem70

Contrasting the co-occurrence patterns of NPM1-FLT3-ITD and NPM1-RAS mouse acute myeloid leukemias by whole exome sequencing.. Molecular Synergy in NPM1 AML

急性髓系白血病基因组学

突变协同效应分析

Mutations affecting NPM1 define the commonest subgroup of acute myeloid leukemia (AML). They frequently co-occur with mutations of FLT3, usually internal tandem duplications (ITD), and less commonly o

NIAID Data Ecosystem50

Additional file 2 of Relapse-specific genetic patterns in the exomic mutational landscape in acute myeloid leukemia

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