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OCA-B/Pou2af1 is sufficient to promote CD4 memory and prospectively identifies memory precursors

NIAID Data Ecosystem2026-05-01 收录

CD4记忆T细胞分化

免疫记忆调控

数据链接：

https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE214310 数据链接链接失效反馈

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资源简介：

This SuperSeries is composed of the SubSeries listed below. Refer to individual Series

应用场景：

创建时间：

2024-02-29

免疫记忆调控

B细胞与T细胞互作

The presence of B cells is essential for the formation of CD8 T cell memory after infection and vaccination. In this study, we investigated whether B cells influence the programming of naïve CD8 T cel

NIAID Data Ecosystem70

mRNA-seq of Tgfbr2 KO CD8 T cells (d30). mRNA-seq of Tgfbr2 KO CD8 T cells (d30)

CD8T细胞分化转录组

免疫记忆调控

This experiment was performed to examine transcriptional differences between differentiated (Tim3+) and stem-like (CXCR5+) WT vs. Tgfbr2 KO antigen-specific CD8 cells at day 30 after LCMV clone 13 inf

NIAID Data Ecosystem50

Expression data from sorted Id3-GFP hi Id2-YFP int and Id3-GFP lo Id2-YFP hi activated CD8 T cells

CD8T细胞分化

免疫记忆调控

During an immune response, CD8 T cells fall along a gradient of memory potential, but the regulators of these fate decsisions are not well understood. We utlized Id3-GFP and Id2-YFP reporter mice to e

NIAID Data Ecosystem30

Single-cell transcriptomics reveals core regulatory programs that determine the heterogeneity of circulating and tissue-resident memory CD8+ T cells

单细胞转录组学

免疫记忆调控

This SuperSeries is composed of the SubSeries listed below. Refer to individual Series

NIAID Data Ecosystem60

PI3Kd coordinates transcriptional, epigenetic and metabolic changes to promote effector CD8+ T cells differentiation at the expense of memory.

T细胞分化

免疫记忆调控

Patients with Activated-PI3Kd Syndrome (APDS) present with sinopulmonary infections, lymphadenopathy and CMV and/or EBV viremia, yet why patients fail to clear certain viral infections remains incompl

NIAID Data Ecosystem40

OCA-B/Pou2af1 is sufficient to promote CD4 memory and prospectively identifies memory precursors

资源简介：

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