E-PROT-122

NIAID Data Ecosystem2026-05-01 收录

杜氏肌营养不良症

体细胞基因编辑治疗

数据链接：

https://www.omicsdi.org/dataset/atlas-experiments/E-PROT-122 数据链接链接失效反馈

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资源简介：

Proteome analysis of skeletal muscle samples from a porcine model of Duchenne muscular dystrophy treated by somatic gene editing

创建时间：

2024-02-20

Molecular hydrogen alleviates motor deficits and muscle degeneration in mdx mice

杜氏肌营养不良症

分子氢治疗

Objective : Duchenne muscular dystrophy (DMD) is a devastating muscle disease caused by a mutation in DMD encoding dystrophin. Oxidative stress accounts for dystrophic muscle pathologies in DMD.

DataCite Commons2020-09-03 更新60

dataset for old mdx mice with BLS-M22

杜氏肌营养不良症

药物疗效评估

This dataset includes all the experimental data for old mdx mice treated with BLS-M22.

Mendeley Data40

Genetic Modifiers of Duchenne Muscular Dystrophy and Dilated Cardiomyopathy

杜氏肌营养不良症

扩张型心肌病风险分层

ObjectiveDilated cardiomyopathy (DCM) is a major complication and leading cause of death in Duchenne muscular dystrophy (DMD). DCM onset is variable, suggesting modifier effects of genetic or environm

Figshare2016-01-15 更新20

scRNAseq RAT DIA WT and DMD 12months

单细胞转录组学

杜氏肌营养不良症

Duchenne muscular dystrophy (DMD) is a fatal muscle-wasting disorder caused by mutations in the Dystrophin gene with no therapeutic option. To bridge the gap between preclinical and therapeutic evalua

NIAID Data Ecosystem40

Patient’s NSAA peak visit in the whole population and subgroups divided by brain dystrophin involvement and corticosteroids duration.

神经肌肉疾病

杜氏肌营养不良症

Patient’s NSAA peak visit in the whole population and subgroups divided by brain dystrophin involvement and corticosteroids duration.

Figshare2022-07-29 更新40