首页/数据集/Lithium promotes longevity via GSK-3 and NRF-2 and is additive to other anti-aging interventions

Lithium promotes longevity via GSK-3 and NRF-2 and is additive to other anti-aging interventions

ChEBI2016-04-07 更新2026-07-12 收录

衰老生物学

药物机制研究

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-MTAB-3809 数据链接链接失效反馈

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资源简介：

Lithium promotes longevity via GSK-3 and NRF-2 and is additive to other anti-aging interventions

应用场景：

创建时间：

2016-04-07

衰老生物学

能量代谢调控

Aging leads to a gradual decline in physical activity and disrupted energy homeostasis. The NAD+-dependent SIRT6 deacylase regulates aging and metabolism through mechanisms that largely remain unknown

NIAID Data Ecosystem90

衰老生物学

代谢基因调控

In living organisms, ageing is widely considered to be the result of a multifaceted process consisting of the progressive accumulation of damage over time, having implications both in terms of functio

Figshare2016-08-13 更新60

The Chemotherapeutic Agent, 5,6-Dimethylxanthenone-4-Acetic Acid,. Mus musculus

药物机制研究

信号通路

Vascular disrupting agents (VDA) represent a novel approach to the treatment of cancer, resulting in collapse of tumor vasculature and tumor death. 5,6-Dimethylxanthenone-4-acetic acid (DMXAA) is a VD

NIAID Data Ecosystem50

Top three gene ontology biological processes resulting from the analysis of differential regulated genes between antioxidant drugs and the respective solvent controls.

基因本体富集分析

药物机制研究

This analysis was performed using the DAVID program (see MM). Count indicates the number of genes belonging to the indicated gene ontology biological processes that we found regulated by a factor 2; %

NIAID Data Ecosystem60

Senescence in the city: exploring ageing patterns of a longâlived raptor across an urban gradient

城市鸟类生态

衰老生物学

In many vertebrates, productivity and survival usually increase with age and then start to decline above a certain age; processes known as reproductive and actuarial senescence. Senescence is widely b

DataONE2019-10-10 更新50

Lithium promotes longevity via GSK-3 and NRF-2 and is additive to other anti-aging interventions

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