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Table S6 from ZNF397 Deficiency Triggers TET2-Driven Lineage Plasticity and AR-Targeted Therapy Resistance in Prostate Cancer

NIAID Data Ecosystem2026-05-02 收录

前列腺癌治疗耐药

谱系可塑性

数据链接：

https://figshare.com/articles/dataset/Table_S6_from_ZNF397_Deficiency_Triggers_TET2-Driven_Lineage_Plasticity_and_AR-Targeted_Therapy_Resistance_in_Prostate_Cancer/26466943 数据链接链接失效反馈

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资源简介：

Table S6. Key Resources

应用场景：

创建时间：

2024-08-02

m6ARNA修饰功能

前列腺癌治疗耐药

Genomic and transcriptomic alterations are insufficient to explain the variance in protein expression seen in cancer. Recent evidence has highlighted the role of N6-methyladenosine (m6A) in the regula

NIAID Data Ecosystem60

RB1 loss promotes a noncanonical activator function of LSD1 and sensitizes castration-resistant prostate cancer to LSD1 inhibition [ChIP-seq]

去势抵抗性前列腺癌

谱系可塑性

Genomic loss of RB1 is a common alteration in castration-resistant prostate cancer (CRPC) and is associated with poor patient outcomes. RB1-loss is also a driver event that promotes the neuroendocrine

NIAID Data Ecosystem20

Emerin Dysregulation Drives the Very-Small-Nuclear Phenotype and Lineage Plasticity that Associates with a Clinically Aggressive Subtype of Prostate Cancer

前列腺癌侵袭性亚型

谱系可塑性

Background: Circulating tumor cells (CTCs) with a very-small-nuclear phenotype (vsnCTCs) in prostate cancer (PCa) are characterized by nuclei smaller than 8.5 μm. Our previous studies established an a

NIAID Data Ecosystem10

Transdifferentiation as a mechanism of treatment resistance in a mouse model of castration-resistant prostate cancer

前列腺癌治疗耐药

转分化机制

Analysis of transcriptome of prostate tissue from the anterior lobe or tumor from 9, 12, 13, 14, and 16 months old mouse Prostate tissue or tumor from 9 month old Nkx3.1CreERT2/+ mice, 14 month old Nk

NIAID Data Ecosystem30

Single-cell RNA-seq reveals a subpopulation of prostate cancer cells with enhanced cell cycle-related transcription and attenuated androgen response

单细胞转录组学

前列腺癌治疗耐药

Increasing evidence indicates that minor subpopulations intrinsic to androgen-independence are present in prostate cancer cells, poised to become clonal dominance under prolonged androgen-deprivation

NIAID Data Ecosystem10

Table S6 from ZNF397 Deficiency Triggers TET2-Driven Lineage Plasticity and AR-Targeted Therapy Resistance in Prostate Cancer

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