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Additional file 2 of PTEN mutant non-small cell lung cancer require ATM to suppress pro-apoptotic signalling and evade radiotherapy

Figshare2022-04-27 更新2026-04-28 收录

肿瘤放射抵抗机制

癌基因功能

数据链接：

https://figshare.com/articles/dataset/Additional_file_2_of_PTEN_mutant_non-small_cell_lung_cancer_require_ATM_to_suppress_pro-apoptotic_signalling_and_evade_radiotherapy/19670209 数据链接链接失效反馈

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资源简介：

Additional file 2. Table S1 table_SEER_REER-colony_ formation.

应用场景：

创建时间：

2022-04-27

肿瘤放射抵抗机制

DNA损伤修复

BackgroundLung adenocarcinoma (LUAD) is the dominant type of lung neoplasms, and radiotherapy is its mainstay treatment, yet poor prognosis caused by radioresistance remains problematic. Cancer-derive

NIAID Data Ecosystem110

Cyclin K interacts with Ã-catenin to induce Cyclin D1 expression and facilitate tumorigenesis and radioresistance in lung cancer

肿瘤放射抵抗机制

肿瘤生物标志物

Rationale: Radioresistance remains the major cause of local relapse and distant metastasis in lung cancer. However, the underlying molecular mechanisms remain poorly defined. This study aimed to inves

NIAID Data Ecosystem80

Cyclin K interacts with β-catenin to induce Cyclin D1 expression and facilitate tumorigenesis and radioresistance in lung cancer. Cyclin K interacts with β-catenin to induce Cyclin D1 expression and facilitate tumorigenesis and radioresistance in lung cancer

肿瘤放射抵抗机制

CyclinK/β-catenin通路

NIAID Data Ecosystem60

Cyclin K interacts with β-catenin to induce Cyclin D1 expression and facilitate tumorigenesis and radioresistance in lung cancer

肿瘤放射抵抗机制

肿瘤分子生物学

NIAID Data Ecosystem100

Additional file 9: of Epithelial-mesenchymal transition markers screened in a cell-based model and validated in lung adenocarcinoma

上皮-间质转化标志物

癌基因功能