Gene amplification leading to increased oncogene expression or anticancer drug resistance is a frequent event in cancer. Cytogenetic, genomic, and transcriptomic analyses were used to determine the in

NIAID Data Ecosystem10

Neisseria meningitidis Type IV pilus SB-DATDH variant

原核生物结构生物学

免疫逃逸机制

Neisseria meningitidis Type IV pilus SB-DATDH variant Descriptor: 2,4-bisacetamido-2,4,6-trideoxy-beta-D-glucopyranose, Neisseria meningitidis PilE, SB-DATDH variant, ... Authors: Fernandez-Martinez,

Protein Data Bank Japan2024-10-16 更新00

DataSheet9_Therapeutic Suppression of FAK-AKT Signaling Overcomes Resistance to SHP2 Inhibition in Colorectal Carcinoma.ZIP

化疗耐药机制

AKT信号通路

SHP2 mediates signaling from multiple receptor tyrosine kinases (RTKs) to extracellular signal-regulated kinase (ERK) and Ser and Thr kinase AKT, and its inhibitors offer an unprecedented opportunity

NIAID Data Ecosystem10

PAF1c links S-phase progression to immune evasion and MYC function in pancreatic carcinoma [ChIP-seq/CUT&RUN]

癌症转录调控

免疫逃逸机制

In pancreatic ductal adenocarcinoma (PDAC), endogenous MYC is required for S-phase progression and escape from immune surveillance. Here we show that MYC in PDAC cells is needed for the recruitment of

NIAID Data Ecosystem10

RNA Sequencing following ZNF384 Knockdown

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