Mechanisms of cellular crosstalk in the gastric tumour microenvironment are mediated by YAP1 and STAT3
收藏NIAID Data Ecosystem2026-05-01 收录
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https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE197002
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The association of proinflammatory pathways and activation of the Hippo pathways are largely uncharacterised. In the present study we aimed to assess the functional relationship between Stat3 and Yap1 ablation in a gastric cancer model, the gp130FF mutant mouse in which Yap1 was conditionally deleted with the Tff1 driven-Cre-recombinase. To explore the biological role of YAP1 in gastric cancer we evaluated the transcriptomic profiles of gastric tumor samples obtained from gp130FF and gp130FF;Yap1floxed mice.
创建时间：
2023-12-06
