首页/数据集/Suppression of HSF1 activity by wildtype p53 creates a driving force for p53 loss-of-heterozygosity

Suppression of HSF1 activity by wildtype p53 creates a driving force for p53 loss-of-heterozygosity

ChEBI2021-05-17 更新2026-07-12 收录

肿瘤抑制基因

细胞应激反应

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-MTAB-10416 数据链接链接失效反馈

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Suppression of HSF1 activity by wildtype p53 creates a driving force for p53 loss-of-heterozygosity

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创建时间：

2021-05-17

线粒体生物学

细胞应激反应

Protein- and RNA-rich bodies contribute to the spatial organization of gene expression in the cell and are also sites of quality control critical to cell fitness. In most eukaryotes, mitochondria ha

DataCite Commons2025-06-01 更新130

Wsc1 mutant 4 hour exposure to Congo Red

细胞应激反应

转录组分析

We did transcription profiling on the effect of wsc1 deletion, gene involved in cell wall stress response Keywords: cell wall stress response S. cerevisiae (wsc1 mutant strain BY4741) was grown on YEP

NIAID Data Ecosystem70

WTX overexpression effect on gastric cell line

癌症分子机制

肿瘤抑制基因

We used microarrays to detail the global gene expression in AGS gastric cancer cells overexpressing WTX, a tumor suppressor frequently inactivated in Wilms tumor. Results provide insight into roles fo

NIAID Data Ecosystem30

Novel Elements of the Chondrocyte Stress Response Identified Using an in Vitro Model of Mouse Cartilage Degradation

细胞应激反应

骨关节炎治疗靶点

The destruction of articular cartilage in osteoarthritis involves chondrocyte dysfunction and imbalanced extracellular matrix (ECM) homeostasis. Pro-inflammatory cytokines such as interleukin-1α (IL-1

NIAID Data Ecosystem80

Transcription profiling of transfected A549 lung cancer cell line with new tumor supressor gen ARLTS1 in that shows a very low expression level in comparison with normal lung

肿瘤抑制基因

癌细胞转录组

We have transfected a new tumor suppresor gene named ARLTS1 in A549 lung cancer cell line that shows a very low expression level in comparison with normal lung. This line was transfected using the pMV

NIAID Data Ecosystem50

Suppression of HSF1 activity by wildtype p53 creates a driving force for p53 loss-of-heterozygosity

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