Cell State Dependent Alteration of Melanoma Plasticity and Immunity by the Circadian Transcription Factor Bmal1 [ChIP-Seq]
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https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE202288
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The circadian transcription factor Bmal1 accelerates melanoma tumorigenesis through initial cell state dependent mechanisms in which Hif1??, Sox9 or Myh9, MRTF-SRF, AP-1 are involved and modulate tumor microenvironment to favor tumor growth CHIP-seq of H3K4me3 and H3K27me3 in cells of different Bmal1 status
创建时间：
2023-05-07
