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Strain-dependent Dissociation of Airway Hyper-responsiveness from Inflammation in a House Dust Mite Model of Asthma

ChEBI2010-11-30 更新2026-07-21 收录

哮喘动物模型

气道高反应性机制

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-19223 数据链接链接失效反馈

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资源简介：

Strain-dependent Dissociation of Airway Hyper-responsiveness from Inflammation in a House Dust Mite Model of Asthma

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创建时间：

2010-11-30

哮喘动物模型

气道平滑肌高反应性

Asthma is a heterogeneous disease of the airways characterized by chronic inflammation associated with bronchial and smooth muscle hyperresponsiveness. Currently, different murine models for the study

NIAID Data Ecosystem100

MeRIP Sequencing of Lung Tissues from OVA-induced Acute Allergic Asthma Mice

RNA表观遗传学

哮喘动物模型

The objective of the study was to present a transcriptome-wide m6A methylome profile of lung tissues in mouse model of ovalbumin(OVA)-induced acute allergic asthma. Overall design: We performed MeRIP-

NIAID Data Ecosystem60

Smooth muscle IL-4 receptor activation induces airway hyper-responsiveness

气道高反应性机制

平滑肌基因表达

Selective stimulation of IL-4 receptor on smooth muscle induces airway hyper-responsiveness in mice. Abstract: Production of the cytokines IL-4 and IL-13 is increased in both human asthma and mouse a

NIAID Data Ecosystem50

An Essential Role for Interleukin-5 and Eosinophils in Helminth-Induced Airway Hyperresponsiveness

免疫病理学

气道高反应性机制

Infection with the parasitic helminth Brugia malayi can result in development of a severe asthmatic response termed tropical pulmonary eosinophilia. This disease, thought to result from a host inflamm

PubMed Central60

Overall tracheal stimulations performed at rest and during exercise (electrically induced muscle contractions) for OVA and control rabbits (expressed as absolute value (percentage)).

哮喘动物模型

运动诱导气道反应性

Overall tracheal stimulations performed at rest and during exercise (electrically induced muscle contractions) for OVA and control rabbits (expressed as absolute value (percentage)).

NIAID Data Ecosystem50

Strain-dependent Dissociation of Airway Hyper-responsiveness from Inflammation in a House Dust Mite Model of Asthma

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