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Super-enhancer redistribution as a mechanism of broad gene dysregulation in repeatedly drug-treated cancer cells. Super-enhancer redistribution as a mechanism of broad gene dysregulation in repeatedly drug-treated cancer cells

NIAID Data Ecosystem2026-03-11 收录

癌症表观遗传学

反应机制

数据链接：

https://www.ncbi.nlm.nih.gov/bioproject/PRJNA532526 数据链接链接失效反馈

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资源简介：

This SuperSeries is composed of the SubSeries listed below. Overall design: Refer to individual Series

应用场景：

创建时间：

2019-04-12

单细胞RNA测序

反应机制

We report the application of scRNA sequencing technology for profiling of acetaminophen response in MDMXC462A mice. These data were used to evaluate differential gene expression and changes in cell co

NIAID Data Ecosystem20

DataSheet_1_A Class of Protein-Coding RNAs Binds to Polycomb Repressive Complex 2 and Alters Histone Methylation.pdf

癌症表观遗传学

PRC2-RNA互作

Polycomb repressive complex 2 (PRC2) is a multi-subunit protein complex mediating the methylation of lysine 27 on histone H3 and playing an important role in transcriptional repression during tumorige

NIAID Data Ecosystem20

ALKBH5 impacts malignancy of colorectal cancer via m6A-mediated epigenetic modulation [meRIP-seq]. ALKBH5 impacts malignancy of colorectal cancer via m6A-mediated epigenetic modulation [meRIP-seq]

癌症表观遗传学

RNAm6A修饰

ALKBH5 is one of the important m6A demethylases and is reported to be closely associated with tumorigenesis.In this study, we report gene expression profiles of HCT116 cells treated with ALKBH5 shRNA.

NIAID Data Ecosystem10

Identification of human endogenous retroviruses working as enhancers in cancer cells

癌症表观遗传学

基因调控网络

Human endogenous retroviruses (HERVs) are a sort of transposable elements. HERVs harbor abundant regulatory elements in their sequences and have a potential to work as enhancers modulating the express

NIAID Data Ecosystem20

Genomewide methylation analysis of B16F10 and 4T1 after Decitabine treatment with Reduced Representative Bisulfite Sequencing [RRBS]. Genomewide methylation analysis of B16F10 and 4T1 after Decitabine treatment with Reduced Representative Bisulfite Sequencing [RRBS]

癌症表观遗传学

DNA甲基化分析

We report genomewide methylation levels on CpG sites and segments before and after Decitabine treatment Overall design: Bisulfite treated DNA was built for deep sequencing, in triplicate, using NovaSe

NIAID Data Ecosystem20

Super-enhancer redistribution as a mechanism of broad gene dysregulation in repeatedly drug-treated cancer cells. Super-enhancer redistribution as a mechanism of broad gene dysregulation in repeatedly drug-treated cancer cells

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