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Global gene expression analysis of heterotypic interactions between cancer cells and osteoblasts in vitro

ChEBI2012-01-06 更新2026-07-21 收录

肿瘤-骨细胞互作

骨转移机制

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-29036 数据链接链接失效反馈

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资源简介：

Global gene expression analysis of heterotypic interactions between cancer cells and osteoblasts in vitro

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创建时间：

2012-01-06

骨转移机制

组学靶点发现

BackgroundBone is the most common metastatic site of Breast invasive carcinoma (BRCA). In this study, the bone metastasis-specific regulation network of BRCA was constructed based on prognostic stemne

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Mir-18a mediates osteogenic lesions in bone metastasis of prostate cancer

骨转移机制

成骨分化基因调控

The transcriptome of Mouse embryo osteoblast 3T3-E1 cells was sequenced. The experimental group was 3T3-E1 cells transfected with lentivirus containing mir-18a mimics. The control group was 3T3-E1 cel

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VCAM1 promotes outbreak from dormant bone metastasis

骨转移机制

VCAM1功能研究

A weakly bone metastatic variant of the breast cancer cell line MDA-MB-231, SCP6, gave rise to highly bone metastatic sublines (PD1, PD2A-E) after long time dormancy in vivo. These cell lines were sub

NIAID Data Ecosystem20

PSAT1 drives extracellular vesicle release in multiple types of cancer and promotes bone metastasis by activating osteoclasts. PSAT1 drives extracellular vesicle release in multiple types of cancer and promotes bone metastasis by activating osteoclasts

细胞外囊泡

骨转移机制

Cancer cells secrete extracellular vesicles (EVs) to regulate cells in the tumor microenvironment for their own benefit to grow and survive in the patient’s body. Although emerging evidence has demons

NIAID Data Ecosystem80

Heme Oxygenase-1 (HO-1) Expression in Prostate Cancer Cells Modulates the Oxidative Response in Bone Cells

骨转移机制

骨微环境调控

Prostate cancer (PCa) is a leading cause of death among males. It is currently estimated that inflammatory responses are linked to 15-20% of all deaths from cancer worldwide. PCa is dominated by compl

NIAID Data Ecosystem40

Global gene expression analysis of heterotypic interactions between cancer cells and osteoblasts in vitro

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