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ETS2, ERG promote megakaryopoiesis, synergize with alterations in GATA1 to immortalize hematopoietic progenitor cells

ChEBI2010-06-23 更新2026-07-21 收录

巨核细胞生成

造血祖细胞

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-14506 数据链接链接失效反馈

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ETS2, ERG promote megakaryopoiesis, synergize with alterations in GATA1 to immortalize hematopoietic progenitor cells

应用场景：

创建时间：

2010-06-23

巨核细胞生成

血小板功能基因

Megakaryocytes accumulate mRNA during their maturation, which is required for the correct spatio-temporal production of cytoskeletal proteins, membranes and platelet-specific granules, and for the sub

NIAID Data Ecosystem70

Co-exposure to preadipocytes and Dioxin increase breast cancer cells aggressiveness through the generation of polynuclear giants cells.

癌细胞侵袭

巨核细胞生成

Breast cancer (BC) is an important disease with high incidence as well as mortality among women, and critical socio-economic impacts. In the past 50 years, it has become a major health problem for wom

NIAID Data Ecosystem40

Genome -wide identification of SCL's target genes in Megakaryocyte progenitors

巨核细胞生成

造血转录因子靶基因

Megakaryopoiesis is a complex process that involves major cellular changes and relies on controlled coordination of cellular proliferation and differentiation. These mechanisms are orchestrated in par

NIAID Data Ecosystem30

Population RNA sequencing of Ly6D+ and TN EPLM subpopulations as well as Pro-B cells

造血祖细胞

时序转录组分析

Nowadays, there is an active debate regarding the heterogeneity of apparently phenotypically homogenous progenitors having different multiple lineage potentials. It has been previously characterized a

NIAID Data Ecosystem60

Competing dynamic gene regulatory networks involved in fibroblast reprogramming to haematopoietic progenitor cells [scRNA-Seq]

细胞重编程

造血祖细胞

Direct reprogramming of somatic cells offers a potentially safer therapeutic approach to generate patient-specific haematopoietic cells. However, this strategy is limited by stochasticity of reprogram

NIAID Data Ecosystem30

ETS2, ERG promote megakaryopoiesis, synergize with alterations in GATA1 to immortalize hematopoietic progenitor cells

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