MiR-155 promotes blood-brain barrier dysfunction in neuroinflammation (part 2)

ChEBI2014-04-29 更新2026-07-21 收录

血脑屏障功能障碍

神经免疫调控

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-44693 数据链接链接失效反馈

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资源简介：

MiR-155 promotes blood-brain barrier dysfunction in neuroinflammation (part 2)

创建时间：

2014-04-29

List of upregulated genes.

神经免疫调控

单细胞转录组学

Microglia, the resident immune cells of the brain, are crucial in the development of the nervous system. Recent evidence demonstrates that microglia modulate adult hippocampal neurogenesis by inhibiti

NIAID Data Ecosystem50

Mosaic deletion of claudin-5 reveals rapid non-cell-autonomous consequences of blood-brain barrier leakage

血脑屏障功能障碍

单细胞转录组学

Claudin-5 (CLDN5) is an endothelial tight junction protein essential for blood-brain barrier (BBB) formation. Abnormal CLDN5 expression is common in brain disease, and knockdown of Cldn5 at the BBB ha

NIAID Data Ecosystem10

A population of Regulatory T cells controls nociception through enkephalins to restrain skin inflammation [fixed scRNA-seq]

神经免疫调控

皮肤炎症机制

The skin integrates diverse signals discerned by sensory neurons and immune cells to elicit adaptive responses to a range of stresses. Considering interactions between nervous and immune systems, we q

NIAID Data Ecosystem00

Open chromatin landscape of rat microglia upon inflammatory or pro-invasive polarization

神经免疫调控

染色质可及性分析

Microglia are brain-resident, myeloid cells that play important roles in health and brain pathologies. While data on chromatin accessibility in activated murine microglia are available, there is no su

NIAID Data Ecosystem30

Endothelial cell-derived oxysterol ablation attenuates experimental autoimmune encephalomyelitis

神经免疫调控

实验性自身免疫性脑脊髓炎

The vasculature is a key regulator of leukocyte trafficking into the central nervous system (CNS) during inflammatory diseases including multiple sclerosis. However, the impact of endothelial-derived

NIAID Data Ecosystem00

MiR-155 promotes blood-brain barrier dysfunction in neuroinflammation (part 2)

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