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Tumor gene expression analysis - PI3K Inhibition by Copanlisib in Relapsed or Refractory Indolent Lymphoma

ChEBI2017-12-01 更新2026-07-12 收录

肿瘤基因表达

分子靶向药物

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-MTAB-6088 数据链接链接失效反馈

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资源简介：

Tumor gene expression analysis - PI3K Inhibition by Copanlisib in Relapsed or Refractory Indolent Lymphoma

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创建时间：

2017-12-01

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肿瘤基因表达

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Gene expression profiling of Trop2+ cells in mouse Apc-deficient small intestinal early adenomas. Gene expression profiling of Trop2+ cells in mouse Apc-deficient small intestinal early adenomas

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肿瘤基因表达

黑色素瘤预后标志物

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Structure of SP4160 Bound to IL-2 V69A

分子结构解析

分子靶向药物

Structure of SP4160 Bound to IL-2 V69A Descriptor: 2-GUANIDINO-4-METHYL-PENTANOIC ACID [2-(4-{5-[4-(4-ACETYLAMINO-BENZYLOXY)-2,3-DICHLORO-PHENYL]-2-METHYL-2H-PYRAZOL-3-YL}-PIPERIDIN-1-YL)-2-OXO-ETHYL]

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Expression data of breast tumors in MMTV-PyMT+ female mice treated with DPM or vehicle. Mus musculus

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肿瘤基因表达

Dipyridamole (DPM) is widely used to prevent strokes and vascular thrombosis. Combination therapy of DPM and antimetabolites has shown synergistic anticancer activity. This study investigated the chem

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Tumor gene expression analysis - PI3K Inhibition by Copanlisib in Relapsed or Refractory Indolent Lymphoma

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