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AMPK epidermal inhibition promotes HuR cytoplasmic localization eliciting post-transcriptional inflammatory response in psoriasis

ChEBI2015-09-08 更新2026-07-12 收录

银屑病分子机制

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https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-50598 数据链接链接失效反馈

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AMPK epidermal inhibition promotes HuR cytoplasmic localization eliciting post-transcriptional inflammatory response in psoriasis

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2015-09-08

P2X7受体

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P2X7 is a ligand-gated ion channel which is activated by ATP and displays secondary permeability characteristics. The mechanism of development of the secondary permeability pathway is currently unclea

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Table_6_Genetic analysis of potential biomarkers and therapeutic targets in ferroptosis from psoriasis.xlsx

银屑病分子机制

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IntroductionFerroptosis is associated with multiple pathophysiological processes. Inhibition of ferroptosis has received much concern for some diseases. Nonetheless, there is no study comprehensively

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银屑病分子机制

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To further explore the gene expression signatures in psoriatic, we have employed whole genome microarray expression profiling as a discovery platform to identify the potential genes that play a role i

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Hyperactivation of the IL-17 axis and IL-36 signaling in Card14-mutant pityriasis rubra pilaris model mice. Hyperactivation of the IL-17 axis and IL-36 signaling in Card14-mutant pityriasis rubra pilaris model mice

炎症信号通路

单细胞转录组学

We performed single cell RNA sequencing (scRNA-seq) to further analyze the differentially expressed genes in each cell type from WT and Card14KI mice. As a result of scRNA-seq, changes in gene express

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JNK1 Signaling Downstream of the EGFR Pathway Contributes to Imiquimod-induced Skin Inflammation (RNA-seq)

银屑病分子机制

炎症信号通路

c-Jun N-terminal protein kinase 1 (JNK1) is involved in multiple biological processes but its implication in inflammatory skin diseases is still poorly defined. Herein, we studied the role of JNK1 in

NIAID Data Ecosystem50

AMPK epidermal inhibition promotes HuR cytoplasmic localization eliciting post-transcriptional inflammatory response in psoriasis

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