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MicroRNAs for fine-tuning of mouse Embryonic Stem Cell fate decision through regulation of TGF-Î² signaling

ChEBI2016-02-11 更新2026-07-12 收录

细胞命运调控

TGF-β信号通路

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-76375 数据链接链接失效反馈

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MicroRNAs for fine-tuning of mouse Embryonic Stem Cell fate decision through regulation of TGF-Î² signaling

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创建时间：

2016-02-11

细胞命运调控

毛囊发育分子机制

In adult K14ΔNLef1 mouse, the overexpression of ∆NLef1, a B-catenin dominat negative, in basal keratinocytes leads to the conversion of hair follicles into multilayered epithelial cysts and ectopic se

NIAID Data Ecosystem60

Genetic changes in the transforming growth factor beta (TGF-beta) type II receptor gene in human gastric cancer cells: correlation with sensitivity to growth inhibition by TGF-beta.

癌症分子机制

TGF-β信号通路

We have found several genetic changes in the TGF-beta-type II receptor gene in human gastric cancer cell lines resistant to the growth inhibitory effect of TGF-beta. Southern blot analysis showed dele

PubMed Central1994-09-13 更新40

TGF-β responsive proteins identified by MALDI TOF-TOF.

蛋白质组学分析

TGF-β信号通路

pI theor = theoretical isoelectric point.pI exp = experimental isoelectric point.MW theor = theoretical molecular weight.MW exp = experimental molecular weight.

Figshare2015-12-02 更新30

Transforming Growth Factor β Receptor Type 1 Is Essential for Female Reproductive Tract Integrity and Function

雌性生殖道发育

TGF-β信号通路

The transforming growth factor β (TGFβ) superfamily proteins are principle regulators of numerous biological functions. Although recent studies have gained tremendous insights into this growth factor

Figshare2016-01-18 更新50

Oscillations of the p53-Akt Network: Implications on Cell Survival and Death

细胞命运调控

P53信号通路

Intracellular protein levels of p53 and MDM2 have been shown to oscillate in response to ionizing radiation (IR), but the physiological significance of these oscillations remains unclear. The p53-MDM2

NIAID Data Ecosystem40

MicroRNAs for fine-tuning of mouse Embryonic Stem Cell fate decision through regulation of TGF-Î² signaling

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