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The hijack of a cellular failsafe program at the initiation of colorectal cancers.

ChEBI2021-05-06 更新2026-07-12 收录

结直肠癌发生机制

细胞安全程序劫持

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-MTAB-4090 数据链接链接失效反馈

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The hijack of a cellular failsafe program at the initiation of colorectal cancers.

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创建时间：

2021-05-06

结直肠癌发生机制

DNA复制调控

To examine origin firing upon CDT1 overexpression, EdUseq-HU was performed in HCT116 cells with normal expression levels of CDT1 (OFF) and HCT116 cells with CDT1 overexpression (ON) Overall design: Ce

NIAID Data Ecosystem60

Epithelial deletion of CUL4B increased myeloid-derived suppressor cells-mediated immunosuppression to promote ApcMin/+ tumor formation

肿瘤微环境

结直肠癌发生机制

Cullin 4B (CUL4B) is a scaffold protein of E3 ubiquitin ligase complex and has been found frequently overexpressed in various types of cancer. By repressing tumor suppressors, CUL4B is indicated as pr

NIAID Data Ecosystem40

Telomeric Chromosomal Instability Fosters Paracrine Mediated Cell Competition to Initiate Intestinal Cancer (WGS)

结直肠癌发生机制

染色体不稳定性与肿瘤微环境

Telomere dysfunction drives chromosomal instability (CIN) during the transition from benign adenoma to malignant adenocarcinoma. While CIN provides a mutator mechanism for cancer- relevant genomic eve

NIAID Data Ecosystem30

Lactylation contributes to colorectal carcinogenesis

乳酸化修饰

结直肠癌发生机制

Lactylation contributes to colorectal carcinogenesis

integrated proteome resources50

Critical role for the DNA receptor AIM2 in stem-cell proliferation and cancer

结直肠癌发生机制

肠道细胞生物学

Colorectal cancer is a leading cause of cancer-related deaths. Mutations in the innate immune receptor AIM2 are frequently identified in patients with colorectal cancer, but how AIM2 modulates colonic

NIAID Data Ecosystem40

The hijack of a cellular failsafe program at the initiation of colorectal cancers.

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