Gene expression in lung tissues of CCL11, IL-5 and IFNγ after instillation of 20 nm

NIAID Data Ecosystem2026-03-08 收录

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https://figshare.com/articles/dataset/Gene_expression_in_lung_tissues_of_CCL11_IL_5_and_IFN_after_instillation_of_20_nm/2673385 数据链接链接失效反馈

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Gene expression in lung tissues of CCL11, IL-5 and IFNγ after instillation of 20 nm

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2015-03-06

RNF138 inhibits late inflammatory gene transcription through degradation of SMARCC1 of SWI/SNF complex

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SWI/SNF染色质重塑

As one of the core components of the SWI/SNF complex, SMARCC1 (SAF155, SRG3) plays essential role in activation of the late inflammatory genes in response to microbial challenge. However, little is kn

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Inflammatory program of CF-TG cells transfected with cftr gene.

囊性纤维化基因治疗

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The expression levels of seven commonly regulated genes were determined by qRT-PCR. The results are expressed as the fold changes between CF-TG cells and wt TG cells, and between CF-TG cells transfect

Figshare2015-12-02 更新40

Targeted transcriptomic analysis of C57BL/6 and BALB/c mice during progressive chronic Toxoplasma gondii infection reveals changes in host and parasitic gene expression relating to neuropathology and resolution [Neuroinflammation]. Targeted transcriptomic analysis of C57BL/6 and BALB/c mice during progressive chronic Toxoplasma gondii infection reveals changes in host and parasitic gene expression relating to neuropathology and resolution [Neuroinflammation]

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弓形虫研究

Toxoplasma gondii is a resilient parasite that infects a multitude of warm-blooded hosts and results in a lifelong chronic infection requiring continuous responses by the host. Chronic infection is ch

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Distinct molecular signatures of mild extrinsic and intrinsic atopic dermatitis. Homo sapiens

特应性皮炎分子机制

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Atopic dermatitis (AD) is a common inflammatory skin disease with underlying defects in epidermal function and immune responses. The goal of this study was to investigate differences in gene expressio

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A stimulus-contingent positive feedback loop enables IFN-β dose-dependent activation of pro-inflammatory genes. A stimulus-contingent positive feedback loop enables IFN-β dose-dependent activation of pro-inflammatory genes

干扰素信号通路

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Type I interferons (IFN) induce powerful anti-viral and innate immune responses via the transcription factor, IFN-stimulated gene factor (ISGF3). However, in some pathological contexts type I IFNs are

NIAID Data Ecosystem30

Gene expression in lung tissues of CCL11, IL-5 and IFNγ after instillation of 20 nm

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