RUNX2/CBFB modulates the response to MEK inhibitors through activation of receptor tyrosine kinases in KRAS mutant colorectal cancer
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https://www.ncbi.nlm.nih.gov/sra/SRP226449
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资源简介：
Investigation of the transcriptional consequences of RUNX2 and CBFB loss in the presence and absence of the MEK inhibitor selumetinib in LoVo ERN1 knockout KRAS mutant colorectal cancer cells. Overall design: RNA-seq comparison of LoVo ERN1 KO, LoVo ERN1/RUNX2 double KO, and LoVo ERN1/CBFB double KO cells treated with 1 uM selumetinib or vehicle.
创建时间：
2023-01-11
