首页/数据集/Sex-dependent effects in the aged melanoma tumor microenvironment influence invasion and resistance to targeted therapy. Chhabra et al

Sex-dependent effects in the aged melanoma tumor microenvironment influence invasion and resistance to targeted therapy. Chhabra et al

Name: Sex-dependent effects in the aged melanoma tumor microenvironment influence invasion and resistance to targeted therapy. Chhabra et al
Creator: Johns Hopkins University Bloomberg School of Public Health
Published: 2024-01-01T00:00:00+08:00

Mendeley Data2024-01-01 更新2026-04-09 收录

黑色素瘤肿瘤微环境

靶向治疗耐药性

数据链接：

https://data.mendeley.com/datasets/stdwp2tmy4 数据链接链接失效反馈

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资源简介：

Source western blots

原始蛋白质印迹（western blots）

应用场景：

提供机构：

Johns Hopkins University Bloomberg School of Public Health

创建时间：

2024-01-01

白血病干细胞

靶向治疗耐药性

Cancer driver mutations often show distinct temporal acquisition patterns, but the biological basis for this, if any, remains unknown. RAS mutations occur invariably late in the course of acute myeloi

NIAID Data Ecosystem50

Exosome-mediated interaction in the tumor microenvironment modulates MIR211 response via the DUSP6-ERK5 axis and contributes to BRAFV600E inhibitor resistance in melanoma

肿瘤微环境

靶向治疗耐药性

It is uncertain whether the microRNA MIR211 is a tumor suppressor or an oncogene. To resolve this, we ectopically expressed MIR211 in BRAFV600E-mutant A375 melanoma cells and examined its effect in mo

NIAID Data Ecosystem30

SLAMF1/CD150 regulates chemotaxis, autophagy and therapy responses in chronic lymphocytic leukemia patients

白血病分子机制

靶向治疗耐药性

This work shows that signaling-lymphocytic-activation-molecule-1 (SLAMF1), a co-stimulatory molecule and a microbial sensor, is expressed by normal CD19+/CD5+ B-lymphocytes. Its expression is lost in

NIAID Data Ecosystem20

Support data_ FOXD1 Promotes Dedifferentiation and Targeted Therapy Resistance in Melanoma by Regulating the Expression of CTGF.zip

黑色素瘤

靶向治疗耐药性

In this manuscript, we show that FOXD1 is highly expressed in melanoma cells and is associated with a poor survival in patients with metastatic melanoma. FOXD1 can promote melanoma cells resistance to

NIAID Data Ecosystem30

Origins of BRAF-mutant hematologic malignancies and their therapeutic resistance

恶性肿瘤分子机制

靶向治疗耐药性

The Mitogen-Activated Protein Kinase (MAPK) signaling pathway has been critically important in understanding pathology and developing targeted therapy for many cancers, but high frequency mutations in

NIAID Data Ecosystem20

Sex-dependent effects in the aged melanoma tumor microenvironment influence invasion and resistance to targeted therapy. Chhabra et al

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