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Additional file 2 of Expression and significance of Fractalkine/CX3CL1 in MPO-AAV-associated glomerulonephritis rats

NIAID Data Ecosystem2026-05-02 收录

肾小球肾炎动物模型

血管炎症机制

数据链接：

https://figshare.com/articles/dataset/Additional_file_2_of_Expression_and_significance_of_Fractalkine_CX3CL1_in_MPO-AAV-associated_glomerulonephritis_rats/26122865 数据链接链接失效反馈

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资源简介：

Supplementary Material 2

应用场景：

创建时间：

2024-06-27

血管炎症机制

脂蛋白信号通路

High density lipoprotein has anti-inflammatory effects in addition to mediating reverse cholesterol transport. While many of the chronic anti-inflammatory effects of high density lipoprotein (HDL) are

NIAID Data Ecosystem90

Aldosterone induces trained immunity: the role of fatty acid synthesis.

免疫代谢

血管炎症机制

Supranormal levels of aldosterone are associated with increased cardiovascular risk in humans, and with accelerated atherosclerosis in animal models. Atherosclerosis is a low-grade inflammatory disord

NIAID Data Ecosystem50

EETs Attenuate Ox-LDL-Induced LTB4 Production and Activity by Inhibiting p38 MAPK Phosphorylation and 5-LO/BLT1 Receptor Expression in Rat Pulmonary Arterial Endothelial Cells

血管炎症机制

脂质介质信号通路

Cytochrome P-450 epoxygenase (EPOX)-derived epoxyeicosatrienoic acids (EETs), 5-lipoxygenase (5-LO), and leukotriene B4 (LTB4), the product of 5-LO, all play a pivotal role in the vascular inflammator

Figshare2016-01-15 更新30

The RNA sequence of descending aorta (DA) in both amotl2ec+/ec+ and amotl2ec-/ec- mice

内皮细胞力学转导

血管炎症机制

EC alignment and cytoskeletal reorganization in response to laminar blood flow is protective against inflammation. The lack of alignment of amotl2ec-/ec- EC in the aorta raised the question whether th

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Gene expression of HUVECS treated with either siRNA to CTRL, PDHA1, or SREBF2 and treated with TNF 16 hours. Gene expression of HUVECS treated with either siRNA to CTRL, PDHA1, or SREBF2 and treated with TNF 16 hours

血管炎症机制

代谢调控基因

The endothelium is a major target of the proinflammatory cytokine, tumor necrosis factor alpha (TNFα). Exposure of endothelial cells (EC) to proinflammatory stimuli leads to an increase in mitochondri

NIAID Data Ecosystem50

Additional file 2 of Expression and significance of Fractalkine/CX3CL1 in MPO-AAV-associated glomerulonephritis rats

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