HIP1 mediates prostate carcinogenesis and progression through STAT3 signalling

ChEBI2016-09-01 更新2026-07-12 收录

前列腺癌分子机制

STAT3信号通路

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HIP1 mediates prostate carcinogenesis and progression through STAT3 signalling

创建时间：

2016-09-01

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前列腺癌分子机制

基因表达调控

The goals of this study is to find effect of overexpression-TFEB in prostate cancer cell (DU145 cell line). Overall design: To further explore the potential molecular mechanisms of how TFEN affects ca

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The biological role of PSMA, a biomarker expressed in prostate cancer and by the neovasculature of solid tumors, remains unknown. Although PSMA processes folates in our duodenum, and in the brain clea

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ERG overexpression is the most frequent molecular alteration in prostate cancer. We analyzed different stages of prostate cancer to identify genes that were coexpressed with ERG overexpression. In pri

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Constitutively Active Androgen Receptor Variants Upregulate Expression of Mesenchymal Markers in Prostate Cancer Cells

前列腺癌分子机制

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Androgen receptor (AR) signaling pathway remains the foremost target of novel therapeutics for castration-resistant prostate cancer (CRPC). However, the expression of constitutively active AR variants

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Gene expression profiles regulated by Stat5a/b vs. Stat3 in human prostate cancer cells.

前列腺癌分子机制

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Identification of the molecular changes that promote viability and metastatic behavior of prostate cancer cells is critical for the development of improved therapeutic interventions for prostate cance

NIAID Data Ecosystem50

HIP1 mediates prostate carcinogenesis and progression through STAT3 signalling

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