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资源简介:
Primary TNBC tumor in the presence or absence of SHP2
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创建时间:
2025-01-24
相关数据集
Genomic Regulation of Invasion by STAT3 in Triple Negative Breast Cancer
Breast cancer is a heterogeneous disease comprised of four molecular subtypes defined by whether the tumor-originating cells are luminal or basal epithelial cells. Breast cancers arising from the lumi
NIAID Data Ecosystem70
SMARCA4 loss-of-function induces cisplatin resistance to activate YAP1-dependent epithelial-to-mesenchymal in triple negative breast cancer
We report transcriptome profiling (RNA-seq) for SMARA4 knock-out to elucidate the association with cisplatin-resistance and SMARCA4 loss-of-function using triple negative breast cancer cell lines. Ove
NIAID Data Ecosystem50
Hazard ratios (95%CI) for 22,812 white, black, Hispanic, and Asian/Pacific Islander women with triple negative breast cancer.
Hazard ratios are adjusted for age, stage, grade, socioeconomic status, and treatment.
Figshare2018-04-27 更新00
ChIP-seq of E4F1 in SUM159 triple negative breast cancer cell line
E4F1 is a ubiquitously expressed zinc-finger protein of the Gli-Kruppel family that was first identified, more than 30 years ago, as a cellular target of the adenoviral oncoprotein E1A13S (Ad type V),
NIAID Data Ecosystem60
PSAT1 gene as a biomarker for targeting triple negative breast cancer in presence of Rapamycin
The Triple-Negative Breast Cancers (TNBCs) poor survival outcomes are related to not responding to hormonal therapy or targeting HER2 protein. The aim of this study was to introduce a drug among the c
Taylor & Francis Group2025-10-21 更新10



