Study of nuclear actin-dependent Meg3 expression

This study investigates nuclear actin-dependent Meg3 expression suppression of metabolic genes by affecting promoter and promoter-enhancer interactions at sites of elevated H3K27 acetylation levels本研究探究了依赖核肌动蛋白（nuclear actin）的Meg3（母系表达基因3）通过在H3K27乙酰化（H3K27 acetylation）水平升高的位点影响启动子与启动子-增强子相互作用，进而抑制代谢基因表达的相关机制。