Loss of ATM accelerates pancreatic cancer formation and epithelial–mesenchymal transition

ChEBI2015-08-07 更新2026-07-12 收录

癌症分子机制

上皮-间质转化

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https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-68808 数据链接链接失效反馈

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Loss of ATM accelerates pancreatic cancer formation and epithelial–mesenchymal transition

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2015-08-07

Additional file 3 of ALKBH5 prevents hepatocellular carcinoma progression by post-transcriptional inhibition of PAQR4 in an m6A dependent manner

癌症分子机制

依赖性基因调控

Additional file 3. Overexpression of ALKBH5 decreased the proliferation capacity in 2 HCC cell lines, while co-overexpressed of PAQR4 reverted this phenomenon (A and B). Overexpression of ALKBH5 decre

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DataSheet4_PRR11 promotes cell proliferation by regulating PTTG1 through interacting with E2F1 transcription factor in pan-cancer.zip

癌症分子机制

细胞周期调控

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癌症分子机制

小檗碱药理作用

The human gastric cancer cell line SGC-7901 was treated by berberine at 20uM for 24h, and the control group was not treated with berberine. Total RNA was extracted, and sequenced. The mRNA and miRNA e

Figshare2017-12-28 更新20

WTX overexpression effect on gastric cell line

癌症分子机制

肿瘤抑制基因

We used microarrays to detail the global gene expression in AGS gastric cancer cells overexpressing WTX, a tumor suppressor frequently inactivated in Wilms tumor. Results provide insight into roles fo

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A renal cancer-associated, renal tubule-specific, HIF-binding enhancer of oncogenic MYC and PVT1 expression. Homo sapiens

癌症分子机制

MYC转录因子

Clear cell renal cell carcinoma is characterized by loss of function of the von Hippel-Lindau tumor suppressor gene (VHL) and unrestrained activation of hypoxia inducible transcription factors (HIF).

NIAID Data Ecosystem30

Loss of ATM accelerates pancreatic cancer formation and epithelial–mesenchymal transition

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