Understanding the molecular mechanism of herpes simplex virus type 1 (HSV-1) latent infection and reactivation in neurons requires the use of model systems. However, establishing a quiescent infection
A fundamental question in herpes simplex virus (HSV) pathogenesis is the consequence of viral reactivation to the neuron. Evidence supporting both post-reactivation survival and demise is published. T
3A-B. Neurons were latently infected with HSV-1 Stayput-GFP and reactivated in the presence of cycloheximide (CHX; 10 μg/ml) added at either the same time as LY294002 or 10 hours later. 3A. Relati
The possibility of HIV-1 eradication has been limited by the existence of latently infected cellular reservoirs. Studies to examine control of HIV latency and potential reactivation have been hindered
The reactivation of various viruses following COVID-19 infection has been reported in several studies. The impact of SARS-CoV-2 infection on the reactivation of other viruses remains unclear. The aim
