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Musashi 2 regulates normal hematopoiesis and accelerates leukemogenesis (LK and MS12-inducible)

ChEBI2010-07-21 更新2026-07-21 收录

造血调控

白血病发病机理

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-22773 数据链接链接失效反馈

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资源简介：

Musashi 2 regulates normal hematopoiesis and accelerates leukemogenesis (LK and MS12-inducible)

应用场景：

创建时间：

2010-07-21

miRNA功能

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Expression of the mirn23a cluster in murine hematopoietic progenitors blocks B cell development. Overexpressed individual cluster members miRs-23a, 24-2, and -27a in 70/3 mouse pre-B cells in order to

NIAID Data Ecosystem60

Genome-scale analysis in blood progenitor and mast cells reveals how lineage-affiliated transcription factors control distinct regulatory programs [Beadchip Experiments #2]

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特异性基因表达

Despite major advances in the generation of genome-wide binding maps, the mechanisms by which transcription factors (TFs) regulate cell type identity have remained largely obscure. Through comparative

NIAID Data Ecosystem40

PHF6 is a key regulator of normal human hematopoiesis by modulating NOTCH1 signaling activity

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Notch信号通路

This SuperSeries is composed of the SubSeries listed below. Refer to individual Series

NIAID Data Ecosystem30

The T-box Transcription Factor Eomesodermin Governs Hemogenic Competence of Yolk Sac Mesodermal Progenitors [scRNA-Seq]

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单细胞转录组学

Extra-embryonic mesoderm (ExM), the earliest cells that traverse through the primitive streak, give rise to the endothelium as well as hematopoietic progenitors in the developing yolk-sac (YS). How a

NIAID Data Ecosystem50

SCL establishes a global repressive environment and cooperates with Polycomb RYBP-PRC1 to suppress alternative lineages in blood-fated cells [ChIP-seq]

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表观遗传学

Understanding the molecular basis of cell fate decision is critical to shed light onto biological processes supporting tissue formation. In particular, whether lineage-fated cells derive from multi-pr

NIAID Data Ecosystem30

Musashi 2 regulates normal hematopoiesis and accelerates leukemogenesis (LK and MS12-inducible)

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