首页/数据集/Supplementary Table S7 from The N6-methyladenosine Epitranscriptomic Landscape of Lung Adenocarcinoma

Supplementary Table S7 from The N6-methyladenosine Epitranscriptomic Landscape of Lung Adenocarcinoma

Figshare2024-11-01 更新2026-04-28 收录

数据链接：

https://figshare.com/articles/dataset/Supplementary_Table_S7_from_The_N_sup_6_sup_-methyladenosine_Epitranscriptomic_Landscape_of_Lung_Adenocarcinoma/27434120 数据链接链接失效反馈

官方服务：

资源简介：

RNA-seq analysis of EML4-overexpressing, EML4-knockdown and control NSCLC cell samples.

应用场景：

创建时间：

2024-11-01

RNA修饰机制

癌症表观转录组学

Additional file 2: Table S2. Duplication events of Catalytic RNA Writer Proteins (RMWs) used in this study.

NIAID Data Ecosystem100

Function and Mechanism of METTL5-mediated 18S rRNA m6A1832 modification in hepatocellular carcinoma. Function and Mechanism of METTL5-mediated 18S rRNA m6A1832 modification in hepatocellular carcinoma

癌症表观转录组学

翻译调控机制

Tumor recurrence is main pattern of treatment failure for early-stage hepatocellular carcinoma (HCC). However, the molecular mechanisms underlying disease recurrence remain poorly understood. Here, we

NIAID Data Ecosystem80

Comparison of tumor-associated YAP1 fusions identifies a recurrent set of functions critical for oncogenesis [mouse RNA-seq]. Comparison of tumor-associated YAP1 fusions identifies a recurrent set of functions critical for oncogenesis [mouse RNA-seq]

YAP1融合致癌机制

肿瘤RNA-seq分析

Several different YAP1 gene fusions have been identified in various human cancers. We performed RNA sequencing on human neural stem cells overexpressing either YAP1-MAMLD1, YAP1-FAM118B, or YAP1-TFE3,

NIAID Data Ecosystem30

MUC1-C IS A MASTER REGULATOR OF SMALL CELL LUNG CANCER PROGRESSION [RNA-seq]. MUC1-C IS A MASTER REGULATOR OF SMALL CELL LUNG CANCER PROGRESSION [RNA-seq]

小细胞肺癌分子机制

肿瘤RNA-seq分析

Small cell lung cancer (SCLC) is a recalcitrant malignancy associated with dysregulation of MYC signaling and the CDK-RB-E2F axis. We report that expression of the oncogenic MUC1-C protein in SCLC cel

NIAID Data Ecosystem90

WDR5 facilitates cholangiocarcinoma metastasis and EMT by promoting HIF-1α accumulation via directly interaction with c-Myc and HDAC2-PHD2 axis

胆管癌转移机制

肿瘤RNA-seq分析

WDR5 was an important co-factor for c-Myc-induced transcriptional activation and malignant progression. We analyzed the differentially expressed genes in scramble and shWDR5 QBC-939 cells. QBC-939 ce

NIAID Data Ecosystem50

Supplementary Table S7 from The N6-methyladenosine Epitranscriptomic Landscape of Lung Adenocarcinoma

资源简介：

相关数据集