Loss of the epithelial adhesion molecule E-cadherin is thought to enable metastasis by disrupting intercellular contacts - an early step in metastatic dissemination. To further investigate the molecu

NIAID Data Ecosystem60

NCOA5 knockout suppresses epithelial-to-mesenchymal transition (EMT) impairing cell proliferation and migration in Hepatocellular Carcinoma cells. NCOA5 knockout suppresses epithelial-to-mesenchymal transition (EMT) impairing cell proliferation and migration in Hepatocellular Carcinoma cells

肝细胞癌机制研究

上皮-间质转化（EMT）

Nuclear receptor coactivator 5 (NCOA5) is an AF2-independent coactivator that contains both transcriptional activation and repression domains. Previous studies have shown that NCOA5 plays an important

NIAID Data Ecosystem40

Additional file 2 of Involvement of immune system and Epithelial–Mesenchymal-Transition in increased invasiveness of clustered circulatory tumor cells in breast cancer

乳腺癌循环肿瘤细胞侵袭性

上皮-间质转化（EMT）

Additional file 2. Turquoise (EMT) subnetwork. The list of genes, module membership, the logarithm of fold change, differential analysis. adj-p.value, and gene-significance statistics.

Figshare2021-11-21 更新80

β-catenin activation down-regulates cell-cell junction-related genes and induces epithelial-to-mesenchymal transition in colorectal cancers

结直肠癌转移机制

上皮-间质转化（EMT）

Purpose: WNT signaling activation in colorectal cancers (CRCs) occurs mainly through APC inactivation or, more uncommonly, β-catenin activation. Both processes promote β-catenin nuclear accumulation,

NIAID Data Ecosystem60

Transcription profiling of mouse mammary epithelial cells NMuMG treatd with TGF-b1 - time series experiment

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