Chronic inflammatory response, DNA damage and subsequent genomic instability are hallmarks of carcinogens-induced HCC . We performed RNA-seq analyses of livers from DEN/CCl4-treated Nod2f/f and Nod2-K

NIAID Data Ecosystem30

ABM_Source_结肠隐窝失巢凋亡计算模型模拟数据_存档

生物学建模

肿瘤机制研究

本数据集为结肠隐窝失巢凋亡的基于代理模型（ABM）模拟数据，聚焦结直肠癌（CRC）发生机制研究。模型包含隐窝口腔几何结构精准表征等新特性，模拟再现了细胞死亡局限于隐窝口腔小区域等与体内观察一致的涌现行为，支持探究隐窝细胞数量调控机制及细胞形状假设对生物学行为的影响。论文“An agent-based model of anoikis in the colon crypt displays nov

海数据20

Targeted Deletion of the Metastasis-Associated Phosphatase Ptp4a3 (PRL-3) Suppresses Murine Colon Cancer

癌症基因治疗靶点

肿瘤机制研究

Ptp4a3 (commonly known as PRL-3) is an enigmatic member of the Ptp4a family of prenylated protein tyrosine phosphatases that are highly expressed in many human cancers. Despite strong correlations wit

Figshare2016-01-18 更新50

Chip sequencing after ER stress induced in HCC cells using tunicamycin. Chip sequencing after ER stress induced in HCC cells using tunicamycin

表观遗传调控

肿瘤机制研究

We report the application of the CHIP sequencing-based technology in the high-throughput analysis of histone modifications in HepG2 cells. Endoplasmic reticulum stress related super enhancers were ide

NIAID Data Ecosystem20

The Sp100 component of ND10/PML bodies is a potent tumor suppressor

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