Additional file 1 of The SF3B1R625H mutation promotes prolactinoma tumor progression through aberrant splicing of DLG1

Additional file 1: Table S1. Primers used for qRT-PCR in this study. Table S2. The clinical data of 15 prolactinoma patients. Table S3. siRNA used in this study. Table S4. Antibodies used in this study. Table S5. Differentially expressed gene in Sf3b1 mutant GH3 cells. Table S6. Differentially expressed gene in SF3B1 mutant human prolactinoma. Table S7. Significant alternative splicing events in Sf3b1 mutant GH3 cells. Table S8. Gene Ontology (GO) function of AS genes in Sf3b1 mutant GH3 cells. Table S9. Significant alternative splicing events in SF3B1 mutant human prolactinoma. Table S10. Gene Ontology (GO) function of AS genes in SF3B1 mutant human prolactinoma. Table S11. Gene Ontology (GO) function of differentially expressed genes in Sf3b1 mutant GH3 cells. Table S12. KEGG pathway analysis of differentially expressed genes in Sf3b1 mutant GH3 cells. Table S13. KEGG pathway analysis of differentially expressed genes in SF3B1 mutant human prolactinoma.