Expression data from HeLa cells with and without human recombinant TIMP-4 treatment. Homo sapiens
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下载链接：
https://www.ncbi.nlm.nih.gov/bioproject/PRJNA259221
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资源简介：
TIMP-4 overexpression increases tumor burden in mice, promotes progenitor cell phenotype and sensitizes cells to apoptosis, by relying on NFkB signaling Overall design: We used microarrays to detail the molecules and signal transduction pathways modulated by hrTIMP-4
创建时间：
2014-08-22
