Streptococcus sanguinis is a commensal member of the oral microbiome involved in opportunistic cardiovascular infections. In the present study, we investigated the contribution of ssa_0094</
CAB2 mutant derived from the clinical isolate RimD2210633. CAB2 lacks TDH hemolysins and its T3SS1 is not functional by means of deletion of exsA, a gene encoding the T3SS1 transcription regulator.
