We characterised genome-wide mutations induced by the O6-dG-methylating agent N-methyl-N'-nitro-N-nitrosoguanidine (MNNG) in mouse embryonic fibroblasts, deficient for the Pol ? catalytic subunit REV3
Here we report our observations that have led us to propose that the transcription elongation factor NusA promotes a novel class of transcription-coupled repair (TCR) in addition to its previously pro