E-MTAB-3101

NIAID Data Ecosystem2026-05-02 收录

成纤维细胞分化

细胞衰老诱导

数据链接：

https://www.omicsdi.org/dataset/atlas-experiments/E-MTAB-3101 数据链接链接失效反馈

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资源简介：

RNA-seq of human foetal foreskin fibroblasts 2 (HFFF2) treated with 2ng/ml TGF-beta-1 to induce myofibroblast differentiation or 10Gy gamma irradiation to induce senescence

创建时间：

2025-01-23

A ‘Synthetic-Sickness’ Screen for Senescence Re-engagement Targets in Mutant Cancer Backgrounds. Homo sapiens

细胞衰老诱导

KRAS突变治疗

Senescence is a universal barrier to immortalisation and tumorigenesis. As such, interest in the use of senescence-induction in a therapeutic context has been gaining momentum in the past few years; h

NIAID Data Ecosystem50

Diversity, Topographic Differentiation, and Positional Memory in Human Fibroblasts

成纤维细胞分化

细胞位置记忆

Diversity, Topographic Differentiation, and Positional Memory in Human Fibroblasts

ChEBI2005-11-02 更新10

Runx1 Promotes Myofibroblast Cell Fate during Development and Fibrosis

成纤维细胞分化

纤维化疾病

Supplemental methods. Supplemental table 1. List of 258 DEGs which are potential direct Runx1 targets.

NIAID Data Ecosystem00

Induction of senescence upon loss of the Ash2l subunit of histone H3K4 methyltransferase complexes (RNA-Seq)

细胞衰老诱导

组蛋白甲基化调控

Post-translational modifications of core histones participate in controlling the expression of genes. Methylation of lysine 4 of histone H3 (H3K4) is associated with open chromatin and gene transcript

NIAID Data Ecosystem20

Identification of epigenetic regulators of fibrotic transformation in cardiac fibroblasts through bulk and single-cell CRISPR screens (ATAC-Seq_2)

核纤层表观遗传调控

成纤维细胞分化

Cardiac fibrosis is mediated by the persistent activity of myofibroblasts, which differentiate from resident cardiac fibroblasts in response to tissue damage and stress signals. The signaling pathways

NIAID Data Ecosystem10

E-MTAB-3101

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