Mice hearts transcriptome sequencing.

NIAID Data Ecosystem2026-05-02 收录

心肌缺血再灌注损伤

心脏转录组表达谱

数据链接：

https://www.ncbi.nlm.nih.gov/bioproject/PRJNA1139944 数据链接链接失效反馈

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资源简介：

Transcriptomic analysis of gene expression changes in the hearts of diabetes-accelerated myocardial ischemia/reperfusion injury mice induced by calponin-1 overexpression.

创建时间：

2024-07-24

Anti-inflammatory and Anti-endoplasmic reticulum stress Effects of catalpol Against myocardial ischemia-reperfusion injury in streptozotocin-induced diabetic rats

心肌缺血再灌注损伤

糖尿病

Abstract The current study was designed to investigate the effects and the mechanism of catalpol on myocardial ischemia-reperfusion (MI/R) injury in a diabetic rat model. Male Sprague-Dawley rats were

DataCite Commons2021-03-24 更新50

DataSheet4_Verapamil Alleviates Myocardial Ischemia/Reperfusion Injury by Attenuating Oxidative Stress via Activation of SIRT1.ZIP

心肌缺血再灌注损伤

SIRT1信号通路药理

Myocardial ischemia/reperfusion (I/R) injury is a potential complication of ischemic heart disease after recanalization. One of the primary reasons for I/R injury is the excessive accumulation of reac

NIAID Data Ecosystem10

RT-qPCR target gene and its primer sequence.

心肌缺血再灌注损伤

细胞焦亡机制

Background Myocardial ischemia-reperfusion injury (I/R) significantly exacerbates cardiomyocyte damage post-recanalization therapy in acute myocardial infarction. Pyroptosis and the NLRP3 inflammasome

NIAID Data Ecosystem10

Data_Sheet_1_Deficiency of a novel lncRNA-HRAT protects against myocardial ischemia reperfusion injury by targeting miR-370-3p/RNF41 pathway.ZIP

心肌缺血再灌注损伤

RNA调控机制

Accumulating evidence indicates that long non-coding RNAs (lncRNAs) contribute to myocardial ischemia/reperfusion (I/R) injury. However, the underlying mechanisms by which lncRNAs modulate myocardial

NIAID Data Ecosystem30

DataSheet12_Propofol Protects Myocardium From Ischemia/Reperfusion Injury by Inhibiting Ferroptosis Through the AKT/p53 Signaling Pathway.ZIP

心肌缺血再灌注损伤

铁死亡信号通路

The molecular mechanism underlying the protective role of propofol against myocardial ischemia/reperfusion (I/R) injury remains poorly understood. Previous studies have shown that ferroptosis is an im

NIAID Data Ecosystem10

Mice hearts transcriptome sequencing.

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