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Candida albicans Clinical Isolates
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创建时间：
2017-09-17
相关数据集
Proteasome-dependent truncation of the negative heterochromatin regulator Epe1 mediates antifungal resistance.
Epe1 histone demethylase restricts H3K9-methylation-dependent heterochromatin, preventing it from spreading over, and silencing, gene-containing regions in fission yeast. External stress induces an ad
NIAID Data Ecosystem80
Candida albicans strain:GZY893 Raw sequence reads. Candida albicans strain:GZY893
This is a highly efficient piggyBac transposon-mediated mutagenesis system using stable haploid C. albicans to conduct genome-wide genetic screens. This study demonstrates the superb power of a new te
NIAID Data Ecosystem50
Directed evolution of the pathogenic mold Aspergillus fumigatus reveals novel triazole-resistance genes
Aspergillus fumigatus is the leading cause of invasive mold infections in immunocompromised patients. Current antifungal treatment primarily depends on the triazole antifungals, which act by noncompet
NIAID Data Ecosystem10
Exploring comparative and transcriptomic analyses to unveil mechanisms of resistance to azoles dependent and independent of CgPdr1 in Candida glabrata clinical isolates
The pathogenic yeast species Candida glabrata has an intrinsically high resilience to azoles and a rapid capability of acquiring resistance. Azole-resistant clinical strains derive mostly from them en
NIAID Data Ecosystem50
Identification of genome wide alternative splicing events in sequential, isogenic clinical isolates of C. albicans reveals a novel mechanism of drug resistance and tolerance to cellular stresses.
Identification of genome wide alternative splicing events in sequential, isogenic clinical isolates of C. albicans reveals a novel mechanism of drug resistance and tolerance to cellular stresses.
NIAID Data Ecosystem20
