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ABCC5 supports osteoclast formation and promotes breast cancer metastasis to bone

ChEBI2012-11-09 更新2026-07-21 收录

乳腺癌骨转移机制

破骨细胞形成调控

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-39494 数据链接链接失效反馈

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ABCC5 supports osteoclast formation and promotes breast cancer metastasis to bone

应用场景：

创建时间：

2012-11-09

乳腺癌骨转移机制

癌细胞基因表达

iRFP713-lebeled iCSCL-10A cells were injected into immunodeficient mice. 5 weeks later, bone metastasized iCSCL-10A cells were extracted from bone marrow was compared with injected cell in vitro cultu

NIAID Data Ecosystem40

Normal human osteoblasts and Hs578t mono and coculture. Homo sapiens

乳腺癌骨转移机制

微环境基因表达

As a model for investigating changes in gene expression in response to epithelial-osteoblast interactions in bone metastases of breast carcinomas, cells that represented malignant epithelial cell comp

NIAID Data Ecosystem30

From breast cancer cell homing to the onset of early bone metastasis: the rol...

乳腺癌骨转移机制

肿瘤微环境

Breast cancer often metastasizes to bone causing osteolytic lesions. Structural and biophysical changes are rarely studied, yet are hypothesized to influence metastatic...

B2FIND20

ABCC5 supports osteoclast formation and promotes breast cancer metastasis to bone. Homo sapiens

乳腺癌分子机制

破骨细胞形成调控

Bone is the primary site of breast cancer metastasis and complications associated with bone metastases can lead to a significantly decreased quality of life in these patients. Thus, it is essential to

NIAID Data Ecosystem40

The IKKβ/FoxO3a axis regulates breast cancer tumorigenesis and bone metastasis. Homo sapiens

乳腺癌骨转移机制

癌症信号通路

IKB Kinase beta (IKKB), a key component of the NFKB signalling pathway plays an important role in inflammation and cancer. Here we describe a previously unknown role of the IKKβ/FoxO3a axis in bone me

NIAID Data Ecosystem20

ABCC5 supports osteoclast formation and promotes breast cancer metastasis to bone

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