Rat ex vivo cataract model using DNA damage inducer

Alliance of Genome Resources2026-08-01 收录

白内障发病机制

DNA损伤诱导白内障

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https://www.ncbi.nlm.nih.gov/gds/?term=GSE194317 数据链接链接失效反馈

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To investigate genes whose expression fluctuates when DNA damage is inflicted and causes lens opacity, microarray analysis was performed.

晶状体研究

白内障发病机制

Age-related breakdown of lenticular crystallins is associated with lenticular disorders such as cataract. Despite playing a critical role in maintaining lens homeostasis, the mechanism(s) and conseque

NIAID Data Ecosystem80

Human Gamma-D crystallin R36S mutant after UV illumination

蛋白质结构解析

白内障发病机制

Human Gamma-D crystallin R36S mutant after UV illumination Descriptor: Gamma-crystallin D Authors: Yorke, B.A, Hill, J.A. Deposit date: 2022-11-16 Release date: 2023-11-29 Last modified: 2026-03-04 Me

Protein Data Bank Japan2026-03-04 更新30

SPARC-null vs. wild-type lens epithelium. Mus musculus

晶状体离子循环调控

白内障发病机制

SPARC is a matricellular glycoprotein involved in regulation of the extracellular matrix, growth factors, adhesion, and migration. SPARC-null mice have altered basement membranes and develop posterior

NIAID Data Ecosystem10

mouse metagenome Raw sequence reads

circRNA表达谱

白内障发病机制

Currently, there is little information on the expression profiles of circRNAs in the lens. CRYBB2 is an abundant protein in the mammalian lens, and its abnormal expression contributes to the developme

NIAID Data Ecosystem50

白内障发病机制

晶状体研究

A large variety of low molecular weight (LMW) peptides, derived from the breakdown of crystallins, have been reported in middle to old age human lenses. The proliferation of these LMW peptides coincid

NIAID Data Ecosystem30

Rat ex vivo cataract model using DNA damage inducer

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