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Evi1 governs Kdm6b-mediated histone demethylation to finely regulate the Laptm4b-driven mTORC pathway in vivo

NIAID Data Ecosystem2026-05-02 收录

组蛋白去甲基化

mTORC信号通路

数据链接：

https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE235442 数据链接链接失效反馈

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This SuperSeries is composed of the SubSeries listed below. Refer to individual Series

应用场景：

创建时间：

2025-01-30

癌症干细胞分化

组蛋白去甲基化

KDM2B is a JmjC domain H3K36me2/H3K36me1 demethylase, which immortalizes cells in culture and contributes to the biology of both embryonic and adult stem and progenitor cells. It also functions as an

NIAID Data Ecosystem70

Histone Demethylation by a Family of UBA Domain-containing Proteins. Histone Demethylation by a Family of UBA Domain-containing Proteins

组蛋白去甲基化

表观遗传调控

Histone methyl groups can be removed by histone demethylases. Although LSD1 and JmjC domain-containing proteins have been identified to be histone demethylases, enzymes responsible for many histone me

NIAID Data Ecosystem60

CpG islands recruit a histone H3 lysine 36 demethylase [Illumina sequencing data]

CpG岛功能机制

组蛋白去甲基化

Eukaryotic gene expression profiles are largely defined by transcription factors that recognize specific DNA sequences in gene regulatory regions and impact RNA polymerase recruitment and transcriptio

NIAID Data Ecosystem60

Crystal structure of histone demethylase kdm2a-h3k36me2 with alpha-kg

组蛋白去甲基化

表观遗传调控

Crystal structure of histone demethylase kdm2a-h3k36me2 with alpha-kg Descriptor: 2-OXOGLUTARIC ACID, Histone H3.2, Lysine-specific demethylase 2A, ... Authors: Cheng, Z.J, Patel, D.J. Deposit date: 2

Protein Data Bank Japan2025-03-26 更新60

Expression data of Jmjd2a and Jmjd2c-regulated genes in plasma cell differentiation. Mus musculus

细胞分化

组蛋白去甲基化

Base on the privious studie from our lab, we found the histone demethylases, Jmjd2a and Jmjd2c are upregulated in stimulated primary B cells, depletion of Jmjd2a and Jmjd2c in stimulated B cells resul

NIAID Data Ecosystem60

Evi1 governs Kdm6b-mediated histone demethylation to finely regulate the Laptm4b-driven mTORC pathway in vivo

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