Lung in neonatal mice: Control vs. transplacental treated with dimethylarsinic acid (DMA). Lung in neonatal mice: Control vs. transplacental treated with dimethylarsinic acid (DMA)

组蛋白H3K9me3修饰

ChIP-seq profiling by Histone H3K9me3 antibody between Control and transplacental treated with 200 ppm dimethylarsinic acid (DMA) for 10 days in lung of neonatal male mice. Overall design: Male and fe

NIAID Data Ecosystem60

ChIP-chip of H3K9me3 in Drosophila embryos at E0-12h time point.

组蛋白H3K9me3修饰

果蝇胚胎早期发育

This is a dataset generated by the Drosophila Regulatory Elements modENCODE Project led by Kevin P. White at the University of Chicago. It contains ChIP-chip data on Affymetrix Drosophila Tiling 2.0R

NIAID Data Ecosystem40

MOESM4 of Decoupling the downstream effects of germline nuclear RNAi reveals that H3K9me3 is dispensable for heritable RNAi and the maintenance of endogenous siRNA-mediated transcriptional silencing in Caenorhabditis elegans

RNAi与转录沉默

H3K9me3组蛋白修饰

Additional file 4: Table S3. RPKM values of 1-kb windows for all ChIP-seq libraries used in this study. GRTS loci, GRH loci, SET-32-dependent H3K9me3, MET-2 SET-25-dependent H3K9me3, and MET-2 SET-25;

NIAID Data Ecosystem20

MOESM11 of Decoupling the downstream effects of germline nuclear RNAi reveals that H3K9me3 is dispensable for heritable RNAi and the maintenance of endogenous siRNA-mediated transcriptional silencing in Caenorhabditis elegans

资源简介：

MOESM11 of Decoupling the downstream effects of germline nuclear RNAi reveals that H3K9me3 is dispensable for heritable RNAi and the maintenance of endogenous siRNA-mediated transcriptional silencing in Caenorhabditis elegans

资源简介：

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