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Complement activation in the peripheral nervous system following the spinal nerve ligation model of neuropathic pain

Alliance of Genome Resources2026-08-01 收录

神经病理机制

补体系统与药物干预

数据链接：

https://www.ncbi.nlm.nih.gov/gds/?term=GSE10238 数据链接链接失效反馈

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Keywords: organ type comparison, disease state analysis and drug treatment

应用场景：

蛋白质异构体功能

神经病理机制

Profilins are small actin binding proteins, which are structurally conserved throughout evolution. They are probably best known to promote and direct actin polymerization. However, they also participa

NIAID Data Ecosystem40

DRG and spinal cord transcriptome

神经病理机制

背根神经节转录组

The dorsal root ganglion transcriptome of rat induced by SNL was analysed in order to study the mechanism of neuropathic pain.

NIAID Data Ecosystem20

Additional file 3 of Calcitonin gene-related peptide regulates spinal microglial activation through the histone H3 lysine 27 trimethylation via enhancer of zeste homolog-2 in rats with neuropathic pain

神经病理机制

表观遗传调控

Additional file 3: Supplementary Table S3. Gene promoters losing H3K27me3 in CGRP treated group.

NIAID Data Ecosystem10

Data_Sheet_2_Gene Transcript Alterations in the Spinal Cord, Anterior Cingulate Cortex, and Amygdala in Mice Following Peripheral Nerve Injury.XLSX

神经病理机制

基因转录组学

Chronic neuropathic pain caused by nerve damage is a most common clinical symptom, often accompanied by anxiety- and depression-like symptoms. Current treatments are very limited at least in part due

NIAID Data Ecosystem30

Supporting data for "Phosphorylated SOX9 Drives Neuroinflammatory Astrocyte Subtypes by Boosting Hexokinase 1 in Neuropathic Pain"

神经病理机制

细胞代谢重编程

Neuropathic pain (NP) is elicited by multiple etiologies, including injuries or diseases, casing spontaneous and persistent pain, which exerts adverse impacts on patients’ quality of life. Currently,

DataCite Commons2024-08-19 更新20

Complement activation in the peripheral nervous system following the spinal nerve ligation model of neuropathic pain

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