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资源简介：
input ChIP-seq data and H3K9me3 ChIP-seq data of atf7ip control & atf7ip mutant
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创建时间：
2022-03-21
相关数据集
Carboplatin-mediated recovery of LPS-induced tolerant macrophages via H3K9me3 modifications
Following the re-exposure to lipopolysaccharide (LPS), macrophages exhibit the immunosuppressive state contribute to low production of proinflammatory cytokines and inability to respond the stimuli, a
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SmcHD1 underlies the formation of H3K9me3 blocks on the inactive X chromosome in mice [ChIP-seq]
Stable silencing of the inactive X chromosome (Xi) in female mammals is crucial for the development of embryos and their postnatal health. SmcHD1 is essential for stable silencing of the Xi, and its f
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ChIP-Seq of FBXO44 and H3K9me3 in MDA-MB-231
FBXO44 bound H3K9me3-modified nucleosomes at the replication fork and recruited the H3K9me3 methyltransferase SUV39H1 for repetitive element silencing post-DNA replication Overall design: 1) ChIP-Seq
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Regulation of histone H3 by the ubiquitin-conjugating enzyme UBE2K determines neurogenesis of human embryonic stem cells [ChIP-Seq]
We find that loss of UBE2K leads to accumulation of histone leading to increase in overall load for epigenetic marks and thus hindering differentiation.Our results provide a novel link between ubiquit
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Genome-wide maps of H3K9me3 with tethered heterochromatin machinery
Purpose: The goal of this study is to characterize the domain(s) of H3K9me3 induced by artifical localization of heterochromatin factors in wild-type and mutant backgrounds. Methods: ChIP-seq was perf
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