Mouse embryonic kidneys (E13.5): HDAC inhibitor (HDACi) treated vs. Control

ChEBI2010-08-01 更新2026-07-21 收录

胚胎肾脏发育

表观遗传药物干预

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https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-19581 数据链接链接失效反馈

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Mouse embryonic kidneys (E13.5): HDAC inhibitor (HDACi) treated vs. Control

创建时间：

2010-08-01

Single cell transcriptional profiling of embryonic mouse nephrogenic zone. Single cell transcriptional profiling of embryonic mouse nephrogenic zone

胚胎肾脏发育

单细胞转录组学

A single-cell RNA-Seq data set was generated using the 10x Genomic platform from E16.5 mice kidney nephrogenic zone cells. Overall design: We isolated and digested embryonic kidneys at E16.5 following

NIAID Data Ecosystem80

Low-dose 5-aza-2′-deoxycytidine protects against early renal injury by increasing klotho expression. Supplementary table

肝肾疾病分子机制

表观遗传药物干预

Supplementary table

Taylor & Francis Group2024-05-16 更新50

An epigenetic vulnerability in PI3K/AKT inhibition resistant cancers is targetable by both bromodomain and HDAC inhibitors

癌细胞耐药性

表观遗传药物干预

Acquisition of resistance to PI3K/AKT-targeted monotherapy regardless of cancer types implies the existence of common mechanisms. Here we demonstrate that while causing glycolysis crisis, acetyl-CoA s

NIAID Data Ecosystem40

Targeting Taxane-Platin Resistant Lung Cancers with JumonjiC Lysine Demethylase Inhibitors (ChIP-Seq)

化疗耐药机制

表观遗传药物干预

Characterization of histone 3 lysine 4 and lysine 27 trimethylation changes upon development of taxane-platin drug resistance in NSCLC cells and evaluation of these histone modifications after treatme

NIAID Data Ecosystem70

HDAC Inhibitors Correct Frataxin Deficiency in a Friedreich Ataxia Mouse Model

弗里德赖希共济失调疾病模型

表观遗传药物干预

BackgroundFriedreich ataxia, an autosomal recessive neurodegenerative and cardiac disease, is caused by abnormally low levels of frataxin, an essential mitochondrial protein. All Friedreich ataxia pat

NIAID Data Ecosystem50

Mouse embryonic kidneys (E13.5): HDAC inhibitor (HDACi) treated vs. Control

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