遇见数据集

JunB is essential for IL-23-dependent pathogenicity of Th17 cells.. Mus musculus

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NIAID Data Ecosystem2026-03-09 收录
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To understand molecular mechanisms by which JunB regulates Th17 differentiation, we performed ChIP-seq analyses of JunB-deficient and control Th17 cells. Overall design: ChIP-Seq analysis of JunB,IRF4,BATF in in vitro generated TH17(β) and TH17(23) cells.

创建时间:
2016-09-07
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