Transcription profiling by array of a mouse model of epileptic tolerance

ChEBI2009-05-22 更新2026-07-21 收录

癫痫模型转录组学

神经病理机制

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Transcription profiling by array of a mouse model of epileptic tolerance

创建时间：

2009-05-22

Mechanistic differences in neuropathic pain modalities revealed by correlating behavior with global expression profiling [microarray]

神经病理机制

背根神经节基因表达

Chronic neuropathic pain is a major morbidity of neural injury, yet its mechanisms are incompletely understood. Hypersensitivity to previously non-noxious stimuli (allodynia) is a common symptom. Here

NIAID Data Ecosystem110

Hippocampus RNA-sequencing of Q808 against PTZ-induced seizure model

癫痫模型转录组学

干预效果评估

The present study aimed to use RNA sequencing to reveal the gene transcriptomic profile of PTZ-induced seizure rats and the difference of the PTZ model rat before and after treatment with Q808. Rats

NIAID Data Ecosystem40

The role of m6A/m-RNA methylation in stress response regulation

表观转录组学

神经病理机制

N6-Methyladenosine (m6A) and N6,2'-O-dimethyladenosine (m6Am) are abundant mRNA modifications that regulate transcript processing and translation. The role of both, here termed m6A/m, in the stress re

NIAID Data Ecosystem30

Transcriptomic profiling in mice with CB1 deletion in primary sensory neurons

神经病理机制

疼痛治疗靶点筛选

in this study, we investigated whether CB1R conditional knockout from peripheral sensory neurons (CB1cKO) in mice may present mechanical allodynia, thermal hyperalgesia and change transcriptome expres

NIAID Data Ecosystem70

Additional file 3 of Positive interaction between GPER and β-alanine in the dorsal root ganglion uncovers potential mechanisms: mediating continuous neuronal sensitization and neuroinflammation responses in neuropathic pain

神经病理机制

神经炎症与神经元敏化

Additional file 3: Table S2. Primers Information of qPCR Experiment.

NIAID Data Ecosystem40