首页/数据集/Table S2 from A Novel miR-146a-POU3F2/SMARCA5 Pathway Regulates Stemness and Therapeutic Response in Glioblastoma

Table S2 from A Novel miR-146a-POU3F2/SMARCA5 Pathway Regulates Stemness and Therapeutic Response in Glioblastoma

Figshare2021-01-01 更新2026-04-28 收录

胶质母细胞瘤分子机制

肿瘤干细胞特性

数据链接：

https://figshare.com/articles/dataset/Table_S2_from_A_Novel_miR-146a-POU3F2_SMARCA5_Pathway_Regulates_Stemness_and_Therapeutic_Response_in_Glioblastoma/22526262 数据链接链接失效反馈

官方服务：

资源简介：

Table S2. Antibodies used in the study

应用场景：

创建时间：

2021-01-01

胶质母细胞瘤分子机制

RNA-seq功能基因组学

Additional file 7: Table S6. RNA-seq analysis of U251 control vs. SERBP1 knockdown samples. Sheet 1, summary of results; sheet 2, list of genes affected by SERBP1 knockdown; sheet 3, list of ncRNA aff

NIAID Data Ecosystem70

Genome-wide analysis upon ZBTB18 expression and/or CTBP2 silencing in Glioblastoma. Genome-wide analysis upon ZBTB18 expression and/or CTBP2 silencing in Glioblastoma

胶质母细胞瘤分子机制

基因调控网络

We have identified the CTBP2 protein as a new ZBTB18 interactor. Here we have evaluated transcriptomic changes upon CTBP2 knockdown in the GBM cell line SNB19. Moreover, we have analyzed whether CTBP2

NIAID Data Ecosystem40

Additional file 6: of Chronophin regulates active vitamin B6 levels and transcriptomic features of glioblastoma cell lines cultured under non-adherent, serum-free conditions

胶质母细胞瘤分子机制

差异表达转录组分析

Table S2. Genes deregulated in NCH644 after CIN knockdown (NGS results). Table of the results from the NGS analysis for NCH644 (comparison CTRL vs. CIN shRNA #1 and #2) including statistics as calcula

NIAID Data Ecosystem30

Additional file 6 of A novel lncRNA MDHDH suppresses glioblastoma multiforme by acting as a scaffold for MDH2 and PSMA1 to regulate NAD+ metabolism and autophagy

胶质母细胞瘤分子机制

lncRNA代谢与自噬调控

Additional file 6.

NIAID Data Ecosystem50

Cellular senescence in malignant cells promotes tumorigenesis in mouse and patient glioblastoma [RNA-seq]. Cellular senescence in malignant cells promotes tumorigenesis in mouse and patient glioblastoma [RNA-seq]

胶质母细胞瘤分子机制

衰老转录组学

We performed a scRNAseq analysis conducted at the endpoint of our GBM mouse model. We compared differentially expressed up-regulated genes in p16Ink4a positive vs p16Ink4a negative malignant cells. Th

NIAID Data Ecosystem30

Table S2 from A Novel miR-146a-POU3F2/SMARCA5 Pathway Regulates Stemness and Therapeutic Response in Glioblastoma

资源简介：

相关数据集